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| 100 |
1 |
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|a Li, Xiaokun.
|0 http://id.loc.gov/authorities/names/n88202051
|1 http://isni.org/isni/0000000381793390.
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| 245 |
1 |
0 |
|a Fibroblast growth factors /
|c Xiaokun Li.
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| 264 |
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1 |
|a London :
|b Academic Press,
|c 2018.
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| 300 |
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|a 1 online resource.
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|a Front Cover; Fibroblast Growth Factors; Fibroblast Growth Factors; Copyright; Contents; Preface; Preface; 1 -- Engineered Growth Factor; Engineered Growth Factors and Cutaneous WoundHealing: Success and Possible Questions in thePast 10 Years; 1. Introduction; 2. Is There a Need for Exogenous Application of Growth Factors in Acute or Chronic Wounds?; 3. Have Growth Factors Altered Clinical Practice in the Past and Will They Alter It in the Future?; 4. Do We Need a Special Delivery System for the Growth Factors in Local Wound Healing?
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| 505 |
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|a 5. Is It Safe to Use Growth Factors to Promote Wound Healing?6. Have All Problems Involved in Wound Healing Been Solved With Growth Factor Application?; 7. Can Perfect Wound Healing Be Achieved in Wounds Treated With Growth Factors?; 8. How Great a Change in Healing Events Would Require a Growth Factor to Become Clinically Important?; 9. What Needs to Be Done to Include Growth Factors in Clinical Practice Within the Next 5-10Years?; References; 2 -- FGFs in Injury Repair and Regeneration.
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| 505 |
8 |
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|a Regulation of Autophagy and Ubiquitinated ProteinAccumulation by bFGF Promotes FunctionalRecovery and Neural Protection in a Rat Model ofSpinal Cord Injury1. Introduction; 2. Materials and Methods; 2.1 Reagents and Antibodies; 2.2 Cell Culture; 2.3 Procedure for Investigating an Animal Model of Spinal Cord Injury; 2.4 Locomotion Recovery Assessment; 2.5 Hematoxylin-Eosin Staining and Nissl Staining; 2.6 Western Blot Analysis; 2.7 Immunofluorescence Staining; 2.8 Statistical Analysis; 3. Results; 3.1 bFGF Decreases Motor Neuron Loss and Improves Functional Recovery of SCI In Vivo.
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|a 3.2 The Protective Effect of bFGF Is Related to Inhibited Autophagy in the SCI Model Mice3.3 Activation of PI3K/Akt/mTOR Signaling Pathways Is Involved in the Role of bFGF in SCI Recovery; 3.4 bFGF Improves Recovery After SCI by Autophagic Clearance of Ubiquitinated Protein Accumulation; 3.5 Combination With the Autophagy Sensitizer Rapamycin Partially Abolishes the Protective Effect of bFGF in SCI Model Rats; 3.6 Exogenous bFGF Protects PC-12 Cells by Inhibition of Excessive Autophagy In Vitro; 4. Discussion; The Antiscar Effects of bFGF on Wound RepairIn Vitro and In Vivo; 1. Introduction.
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|a 2. Materials and Methods2.1 Ethics Statement; 2.2 Animal Model; 2.3 Histological Examination and Immunohistochemistry Staining; 2.4 Terminal Deoxynucleotidyl Transferase-Mediated dUTP Nick-End Labeling Staining; 2.5 Collagen I and Collagen III Quantification; 2.6 Western Blot; 2.7 Fibroblasts Culture; 2.8 Quantitative Real-Time PCR; 2.9 Statistical Analysis; 3. Results; 3.1 bFGF Accelerates Acute Wound Closure in the Rat-Incised Injury Model; 3.2 bFGF Alleviates the Scar Formation in the Rabbit Ear Model; 3.3 Effect of bFGF on Collagen I and Collagen III Synthesis.
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| 588 |
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|a Online resource; title from PDF title page (EBSCO, viewed June 26, 2018)
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|a Fibroblast growth factors.
|0 http://id.loc.gov/authorities/subjects/sh91005352.
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|z Full Text (via ScienceDirect)
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|a 3.4 Effect of bFGF on α-SMA and TGF-β1 Expression.
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